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The expression levels of immune checkpoint molecules were compared across the risk groups and the results demonstrated that TNF superfamily member (TNFSF)18, CD160, adenosine A2a receptor, neuropilin-1, CD200 and TNFSF9 were markedly upregulated, while CD44 was markedly downregulated in the high-risk group compared with the low-risk group (Fig

doi: 10.1007/s12603-013-0378-z

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Bi-allelic mutations in phe-tRNA synthetase associated with a multi-system pulmonary disease support non-translational function

This suggests the reduction in TNF- observed is primarily due to NAC's incorporation

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